A Brief Technical Description
The basis for the Neuralign treatment was part theorized in 1965 by the original ‘Gate Control Theory’ of Melzack and Wall, and thereafter considered, applied and developed in a variety of application methods. During the last 40 years of working with this concept, there have been many development and application stages before resulting in the injury recovery treatment available today that is known as Neuralign and which imparts direct pressure stimulation (manipulation) upon proprioceptors that can inhibit nociceptive receptor activity.
Proprioceptors (nerve receptors) are many and varied. Muscle proprioceptors exist for movement control and limb/joint position detection. For joint movement we have muscle spindles (intrafusal, extrafusal, and specialised fibres) to monitor muscle length whilst afferent nerve endings provide muscle tension information. The golgi tendon organs (afferent nerves) specifically monitor imparted joint muscle forces.
Movement is dependent on proprioceptors that detect the joint displacements during movement. Your sense of touch is possible because of receptors in the skin, and joint sensitivities originate from internally located receptors that provide information about movement and tension in joints, tendons, ligaments and muscles. In this way, proprioceptors, as parts of the central nervous system (CNS) provide kinesthetic information to monitor body movement, balance, joint tension, muscle, tendon and ligament loadings.
The intensity of stimulation upon joint tissues and the muscles influencing a joint will define the degree and duration of ongoing stimulation during joint activity. A resumption or increase of joint activity and frequency will influence the intensity of stimulation. Inhibiting nociceptive receptor activity for this can support a resumption of more normal joint activity, thereby achieving the desired activity frequency.
Remobilization stimulates the muscle
proprioceptors to reduce a pain response and reaction with a change in a muscle
length stimulating the receptors and signaling the CNS monitoring of the variation.